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Archives of Dermatology current issue

ANNUAL REVIEWERS LIST: Our Thanks to the ARCHIVES Peer Reviewers
Mon, 15 Dec 2008 00:00:00 -0000


PubMed: 0001-5555

Skin nodules in rheumatoid arthritis due to infection with Mycobacterium haemophilum.
Seitz CS, Trautemann A, Bröcker EB, Abele-Horn M, Goebeler M Related Articles Skin nodules in rheumatoid arthritis due to infection with Mycobacterium haemophilum. Acta Derm Venereol. 2008;88(4):428-9 Authors: Seitz CS, Trautemann A, Bröcker EB, Abele-Horn M, Goebeler M PMID: 18709331 [PubMed - indexed for MEDLINE]

Latest Issue of American Journal of Clinical Dermatology

Acknowledgment
Adis International Limited,

The American Journal of Dermatopathology - Current Table Of Contents

Eosinophilic Globules in 3 Cases of Glomeruloid Hemangioma of the Head and Neck: A Characteristic Offering More Evidence for Thanatosomes With or Without POEMS.
Page: 539DOI: 10.1097/DAD.0b013e318182c800Authors: Lee, Hwajeong MD; Meier, Frederick A MD; Ma, Chan K MD; Ormsby, Adrian H MD; Lee, Min W MD

Archives of Dermatological Research

Insulin enhances proliferation and viability of human umbilical vein endothelial cells
Tue, 30 Dec 2008 07:07:56 -0000
Abstract  This investigation is a follow-up to our previous in vivo studies revealing that rapid stretch increases tissue insulin in murine skin flaps, coincident with the up-regulation of key angiogenic effectors and enhanced vascularization. In the present study, we used human umbilical vein endothelial cells (HUVECs) as an in vitro model system to determine the role of insulin in the chemical signals regulating the processes of proliferation and viability (survival). MTT-based colorimetric methods demonstrated that insulin enhances proliferation and survival of HUVECs. Western blot analysis revealed that protein kinase B (pAkt [Thr308]) and vascular endothelial growth factor (VEGF) were the insulin-responsive intermediates in proliferating endothelial cells (ECs). In insulin-enhanced survival, both pAkt (Thr308) and pAkt (Ser473) were activated in HUVECs. However, no change in VEGF expression accompanied pAkt activation. The beneficial effects of insulin were abrogated by insulin receptor (IR)/insulin-like growth factor receptor (IGFR) or phosphoinositide-3 kinase (PI3-K) blockade, suggesting that insulin-induced EC proliferation and viability are mediated through pIR/pIGFR and PI3-K effectors. These data provide new insights into the beneficial effects of insulin on vascularization and tissue viability, providing a mechanistic link to the enhancement of healing in acutely stretched skin. Content Type Journal ArticleCategory Original PaperDOI 10.1007/s00403-008-0921-7Authors Carl D. Shrader, West Virginia University Department of Neurobiology and Anatomy, School of Medicine Morgantown WV 26506-9128 USAKelly M. Bailey, West Virginia University Department of Physiology and Pharmacology, Cancer Cell Biology, School of Medicine Morgantown WV 26506-9177 USAGregory W. Konat, West Virginia University Department of Neurobiology and Anatomy, School of Medicine Morgantown WV 26506-9128 USAEugene V. Cilento, West Virginia University Department of Neurobiology and Anatomy, School of Medicine Morgantown WV 26506-9128 USAFrank D. Reilly, West Virginia University Department of Neurobiology and Anatomy, School of Medicine Morgantown WV 26506-9128 USA Journal Archives of Dermatological ResearchOnline ISSN 1432-069XPrint ISSN 0340-3696

BMC Dermatology - Latest articles

Dermatitis associated with exposure to a marine cyanobacterium during recreational water exposure
Nicholas J Osborne and Glen R Shaw Tue, 30 Dec 2008 00:00:00 -0000
Background: Anecdotal evidence reported an outbreak of symptoms on Fraser Island during the late 1990s similar to those expected from exposure to dermotoxins found in the cyanobacterium Lyngbya majuscula. This coincided with the presence of a bloom of L. majuscula. Methods: Records from Fraser Island National Parks First aid station were examined. Information on cyanobacterial blooms at Fraser Island were obtained from Queensland National Parks rangers. Results: Examination of first aid records from Fraser Island revealed an outbreak of symptoms in predominantly in January and February 1998. Conclusions: During a bloom of L. majuscula there were numerous reports of symptoms that could be attributed to dermotoxins found in L. majuscula. The other four years examined had no L. majuscula blooms and the number of L. majuscula symptoms was much reduced. These cases comprised a high percentage of the cases treated at the first aid station.

Dermatology : Last 20 articles

Newsletter No. 4/2009
Mon, 15 Dec 2008 15:57:48 +0100
Dermatology 2009;218:92-96 (DOI:10.1159/000187644)

PubMed: 1167-1122

Type 2 segmental cutaneous leiomyomatosis: an example of mosaicism.
Asín BF, Rosende MB, Gijón RC, Fernández-Guarino M, Olásolo PJ Related Articles Type 2 segmental cutaneous leiomyomatosis: an example of mosaicism. Eur J Dermatol. 2008 Dec 23; Authors: Asín BF, Rosende MB, Gijón RC, Fernández-Guarino M, Olásolo PJ PMID: 19109093 [PubMed - as supplied by publisher]

Exogenous Dermatology : Last 20 articles

Contents Vol. 3, 2004
Mon, 12 Jun 2006 16:18:22 +0100
Exog Dermatol 2004;3:353-354 (DOI:10.1159/000094132)

Expert Reviews: Expert Review of Dermatology: Table of Contents

Role of genetic testing in hereditary melanoma
info@expert-reviews.com (Wendy Kohlmann et al) Tue, 02 Dec 2008 10:39:54 -0000
Expert Review of Dermatology , December 2008, Vol. 3, No. 6, Pages 639-643.

PubMed: 0022-202X

The Role of Interleukin-6 in UVA Protection against UVB-Induced Immunosuppression.
Reeve VE, Tyrrell RM, Allanson M, Domanski D, Blyth L Related Articles The Role of Interleukin-6 in UVA Protection against UVB-Induced Immunosuppression. J Invest Dermatol. 2008 Dec 25; Authors: Reeve VE, Tyrrell RM, Allanson M, Domanski D, Blyth L The proinflammatory cytokine IL-6 is released in the skin following UVB irradiation, but its potential for photoimmune modulation remains unclear. This study utilizes IL-6-deficient mice to demonstrate that IL-6 does not contribute to the normal contact hypersensitivity response, nor to its systemic suppression by UVB radiation or cis-urocanic acid. In contrast, IL-6 was required for the attenuation of UVB- or cis-urocanic acid-induced immunosuppression by sequential or concomitant UVA irradiation. The IL-6 was essential for several reactions previously established to be relevant for UVA photoimmune protection, namely the induction of heme oxygenase-1 (HO-1), the activity of its product carbon monoxide in activating guanylyl cyclase, and the consequent elevation of cutaneous cyclic guanosine monophosphate concentration. In addition, IL-6-deficient mouse skin had an elevated constitutive overexpression of HO activity, apparently not associated with photoimmune protection. This suggested that both the cutaneous level of HO activity, and the receptiveness of the HO-1 gene to stressors like UVA, normally controlled by promoter-binding repressor proteins, may also be under IL-6 control. Thus IL-6 has an important photoimmune protective function through interaction at several levels in the pathway determining the immunologically advantageous actions of UVA radiation. This may constitute a valuable endogenous antiphotocarcinogenic regulatory mechanism.Journal of Investigative Dermatology advance online publication, 25 December 2008; doi:10.1038/jid.2008.377. PMID: 19110542 [PubMed - as supplied by publisher]

Sexually Transmitted Diseases - Current Table Of Contents

Black Women in the United States Decrease Their Number of Recent Sex Partners: Temporal Trends From the National Survey of Family Growth.
Page: 1DOI: 10.1097/OLQ.0b013e3181845b84Authors: Leichliter, Jami S. PhD; Aral, Sevgi O. PhD

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